Now showing items 1-19 of 19

      Deglycosylated MUC1 Ectodomain for the Diagnosis and Treatment of Pseudomonas aeruginosa Lung Infections [1]
      Desialylated Host-Derived MUC1 Ectodomain and Bacterially-Derived Flagellin are Biomarkers for Airway Colonization and Infection with Pseudomonas aeruginosa in Mechanically Ventilated and Cystic Fibrosis Patients [1]
      Expression of MUC1 Mucin Increases Intracellular Calcium Levels Through a CAML-Dependent Mechanism [1]
      Human Airway Epithelia Express Catalytically-Active NEU3 Sialidase [1]
      The Human NEU1-Selective Sialidase Inhibitor, GSC-649, Blocks NEU1-Mediated Bioactivities in Human Airway Epithelia and Lung Microvascular Endothelia [1]
      Intercellular Adhesion Molecules Expressed by Normal, Cystic Fibrosis, and Cancer Airway Epithelial Cell Lines [1]
      Involvement of PI3K, Src, and Grb2 in MUC1 Signaling [1]
      MUC1 Mucin Inhibits Airway Epithelial Barrier Formation Through a beta Catenin Dependent Mechanism [1]
      MUC1 Mucin Inhibits Airway Epithelial Barrier Formation Through a β-Catenin Dependent Mechanism [1]
      NEU1 Sialidase Associates with the MUC1 Cytoplasmic Domain and not the MUC1 Ectodomain [1]
      NEU1 Sialidase Increases Shedding of MUC1 Ectodomain as a Decoy Receptor for Pseudomonas aeruginosa, Decreasing Bacterial Adhesion to Human Airway Epithelia [1]
      NEU1 Sialidase Promotes MUC1 Ectodomain Release from Human Airway Epithelial Cells as a Soluble Decoy Receptor that Blocks Pseudomonas aeruginosa Adhesion [1]
      NEU1 Sialidase Regulates MUC1-Dependent Bacterial Adhesion to and Invasion of Human Airway Epithelia [1]
      Neuraminidase-1 Desialylates the MUC1 Ectodomain to Release a Decoy Receptor that Protects against Lethal Pseudomonas aeruginosa Lung Infection [1]
      Pseudomonas aeruginosa Flagellin Regulates NEU1 Sialidase-Responsive, MUC1 Ectodomain-Dependent Bacterial Adhesion to and Invasion into Human Airway Epithelial Cells [1]
      Pseudomonas aeruginosa Flagellins Induce Interleukin-8 Production in Airway Epithelial Cells [1]
      Pseudomonas aeruginosa Isolates from Multiple Clinical Settings Induce Equal Levels of IL-8 Production by Airway Epithelial Cells [1]
      Respiratory MUC1 Mucin Interacts with Calcium-Modulating Cyclophilin Ligand (CAML) [1]
      Role of Muc1 in Helicobacter pylori Gastric Mucosal Inflammation [1]