Browsing Dr. Erik Lillehoj by Title
Now showing items 1-19 of 19
|Deglycosylated MUC1 Ectodomain for the Diagnosis and Treatment of Pseudomonas aeruginosa Lung Infections |
|Desialylated Host-Derived MUC1 Ectodomain and Bacterially-Derived Flagellin are Biomarkers for Airway Colonization and Infection with Pseudomonas aeruginosa in Mechanically Ventilated and Cystic Fibrosis Patients |
|Expression of MUC1 Mucin Increases Intracellular Calcium Levels Through a CAML-Dependent Mechanism |
|Human Airway Epithelia Express Catalytically-Active NEU3 Sialidase |
|The Human NEU1-Selective Sialidase Inhibitor, GSC-649, Blocks NEU1-Mediated Bioactivities in Human Airway Epithelia and Lung Microvascular Endothelia |
|Intercellular Adhesion Molecules Expressed by Normal, Cystic Fibrosis, and Cancer Airway Epithelial Cell Lines |
|Involvement of PI3K, Src, and Grb2 in MUC1 Signaling |
|MUC1 Mucin Inhibits Airway Epithelial Barrier Formation Through a beta Catenin Dependent Mechanism |
|MUC1 Mucin Inhibits Airway Epithelial Barrier Formation Through a β-Catenin Dependent Mechanism |
|NEU1 Sialidase Associates with the MUC1 Cytoplasmic Domain and not the MUC1 Ectodomain |
|NEU1 Sialidase Increases Shedding of MUC1 Ectodomain as a Decoy Receptor for Pseudomonas aeruginosa, Decreasing Bacterial Adhesion to Human Airway Epithelia |
|NEU1 Sialidase Promotes MUC1 Ectodomain Release from Human Airway Epithelial Cells as a Soluble Decoy Receptor that Blocks Pseudomonas aeruginosa Adhesion |
|NEU1 Sialidase Regulates MUC1-Dependent Bacterial Adhesion to and Invasion of Human Airway Epithelia |
|Neuraminidase-1 Desialylates the MUC1 Ectodomain to Release a Decoy Receptor that Protects against Lethal Pseudomonas aeruginosa Lung Infection |
|Pseudomonas aeruginosa Flagellin Regulates NEU1 Sialidase-Responsive, MUC1 Ectodomain-Dependent Bacterial Adhesion to and Invasion into Human Airway Epithelial Cells |
|Pseudomonas aeruginosa Flagellins Induce Interleukin-8 Production in Airway Epithelial Cells |
|Pseudomonas aeruginosa Isolates from Multiple Clinical Settings Induce Equal Levels of IL-8 Production by Airway Epithelial Cells |
|Respiratory MUC1 Mucin Interacts with Calcium-Modulating Cyclophilin Ligand (CAML) |
|Role of Muc1 in Helicobacter pylori Gastric Mucosal Inflammation |
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