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dc.contributor.authorBen-Josef, E.
dc.contributor.authorWinter, K.
dc.contributor.authorWilliams, T.M.
dc.contributor.authorRegine, W.F.
dc.contributor.authorTimmers, C.D.
dc.contributor.authorRobb, R.
dc.contributor.authorDiPetrillo, T.A.
dc.contributor.authorLeasure, N.C.
dc.contributor.authorMowat, R.
dc.contributor.authorKim, H.
dc.contributor.authorKoenig, J.M.
dc.contributor.authorNarayan, S.
dc.contributor.authorBush, D.A.
dc.contributor.authorFromm, M.
dc.contributor.authorGuha, C.
dc.date.accessioned2022-04-05T18:59:19Z
dc.date.available2022-04-05T18:59:19Z
dc.date.issued2021-11
dc.identifier.urihttp://hdl.handle.net/10713/18468
dc.description.abstractLaboratory experiments show that activation of β-catenin signaling promotes pancreatic cancer (PC) growth, but studies addressing its prognostic value in patients (pts) are scarce and with conflicting results. This question has been examined using samples from a large prospective clinical trial.en_US
dc.description.urihttps://doi.org/10.1016/j.ijrobp.2021.07.328en_US
dc.language.isoenen_US
dc.publisherElsevieren_US
dc.relation.ispartofInternational Journal of Radiation Oncology - Biology - Physicsen_US
dc.rightsCopyright © 2021 Published by Elsevier Inc.en_US
dc.rights.urihttps://www.elsevier.com/tdm/userlicense/1.0/
dc.titleβ-Catenin is Independently Associated With Outcome in Pancreatic Cancer: An Analysis of NRG Oncology/RTOG 9704en_US
dc.typeAbstracten_US
dc.identifier.doi10.1016/j.ijrobp.2021.07.328
dc.source.journaltitleInternational Journal of Radiation Oncology*Biology*Physics
dc.source.volume111
dc.source.issue3
dc.source.beginpageS146
dc.source.endpageS147


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