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dc.contributor.authorAbrams, T.W.
dc.date.accessioned2019-09-19T18:35:43Z
dc.date.available2019-09-19T18:35:43Z
dc.date.issued2017
dc.identifier.urihttps://www.scopus.com/inward/record.uri?eid=2-s2.0-85029875610&doi=10.1016%2fj.cub.2017.07.058&partnerID=40&md5=15170092ed8d7ae4d834f622b8a6b289
dc.identifier.urihttp://hdl.handle.net/10713/10907
dc.description.abstractNew possibilities for treating posttraumatic stress disorder and anxiety disorders involving abnormal memories are emerging from analysis of persistent protein kinase activation and mechanisms of synapse-specific modification, known as synaptic tagging. New possibilities for treating posttraumatic stress disorder and anxiety disorders involving abnormal memories are emerging from analysis of persistent protein kinase activation and mechanisms of synapse-specific modification, known as synaptic tagging.en_US
dc.description.urihttps://doi.org/10.1016/j.cub.2017.07.058en_US
dc.language.isoen_USen_US
dc.publisherCell Pressen_US
dc.relation.ispartofCurrent Biology
dc.subject.meshMemoryen_US
dc.subject.meshNeuronal Plasticityen_US
dc.subject.meshNeuronsen_US
dc.subject.meshSynapsesen_US
dc.titleSynaptic Plasticity: Cleaved Kinases and the Specificity of Erasing Traumatic Memoriesen_US
dc.typeArticleen_US
dc.identifier.doi10.1016/j.cub.2017.07.058


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